Michael V. Cohen MD
Cardiologist | Cardiovascular Disease
2451 Fillingim St Bldg. C Mobile AL, 36617About
Dr. Michael Cohen is a cardiologist practicing in Mobile, AL. Dr. Cohen specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Cohen also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Harvard Med Sch, Boston Ma 1968
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Preconditioning one myocardial region does not neccessarily precondition the whole rabbit heart.
- Xanthine oxidase contributes to preconditioning's preservation of left ventricular developed pressure in isolated rat heart: developed pressure may not be an appropriate end-point for studies of preconditioning.
- The protective and anti-protective effects of ethanol in a myocardial infarct model.
- Mitochondrial K(ATP) channels: role in cardioprotection.
- Acetylcholine leads to free radical production dependent on K(ATP) channels, G(i) proteins, phosphatidylinositol 3-kinase and tyrosine kinase.
- The relative order of mK(ATP) channels, free radicals and p38 MAPK in preconditioning's protective pathway in rat heart.
- Opening of ATP-sensitive potassium channels causes generation of free radicals in vascular smooth muscle cells.
- Dose-response relationships of the protective and antiprotective effects of acute ethanol exposure in isolated rabbit hearts.
- Protection from AMP 579 can be added to that from either cariporide or ischemic preconditioning in ischemic rabbit heart.
- ACh and adenosine activate PI3-kinase in rabbit hearts through transactivation of receptor tyrosine kinases.
- Acetylcholine but not adenosine triggers preconditioning through PI3-kinase and a tyrosine kinase.
- Treatment of orthostatic hypotension.
- Activation of Akt is essential for acetylcholine to trigger generation of oxygen free radicals.
- Mitochondrial K(ATP) channels in preconditioning.
- Acetylcholine-induced production of reactive oxygen species in adult rabbit ventricular myocytes is dependent on phosphatidylinositol 3- and Src-kinase activation and mitochondrial K(ATP) channel opening.
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