Dr. Terri H Finkel M.D.
Rheumatologist (Pediatric) | Pediatric Rheumatology
34th & Civic Center Blvd. Children's Hospital Philadelphia PA, 19104About
Dr. Terri Finkel practices Pediatric Rheumatology in Philadelphia, PA. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Finkel evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Finkel also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Stanford Univ Sch Of Med- Stanford Ca 1982
Board Certification
PediatricsAmerican Board of PediatricsABP- Pediatric Rheumatology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Development and function of autospecific dual TCR+ T lymphocytes.
- Ligation of the CD4 receptor induces activation-independent down-regulation of L-selectin.
- ADP-ribosylation of rho by C3 ribosyltransferase inhibits IL-2 production and sustained calcium influx in activated T cells.
- Potential use of a caspase against HIV.
- Nabumetone induced pseudoporphyria in childhood.
- Biochemical mechanisms of HIV induced T cell apoptosis.
- HIV-1 NL4-3, but not IIIB, inhibits JAK3/STAT5 activation in CD4(+) T cells.
- Ligation of human CD4 interferes with antigen-induced activation of primary T cells.
- Human CD8+ T cells do not require the polarization of lipid rafts for activation and proliferation.
- T cell signaling and apoptosis in HIV disease.
- Chronic GVH prevents anergy in bone marrow self-reactive B cells: a selective increase in post-endoplasmic reticulum processing and trafficking to the cell surface of autoreactive IgM receptors.
- Crosslinking CD4 by human immunodeficiency virus gp120 primes T cells for activation-induced apoptosis.
- CXCR4 engagement is required for HIV-1-induced L-selectin shedding.
- Tracking synapse-associated TCRs.
- Differential gene expression during HIV-1 infection analyzed by suppression subtractive hybridization.
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