Dr. Clement Richard Boland MD
Gastroenterologist | Gastroenterology
3500 Gaston Ave Baylor University Me Dallas TX, 75246About
Dr. Clement Boland is a gastroenterologist practicing in Dallas, TX. Dr. Boland specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Boland performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Gastroenterology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- JC virus DNA is present in the mucosa of the human colon and in colorectal cancers.
- Mismatch repair proficiency and in vitro response to 5-fluorouracil.
- How many mutations does it take to make a tumor?
- Conversion of diploidy to haploidy.
- Steady-state regulation of the human DNA mismatch repair system.
- Molecular genetics of hereditary nonpolyposis colorectal cancer.
- Antisense inhibition of hMLH1 is not sufficient for loss of DNA mismatch repair function in the HCT116+chromosome 3 cell line.
- JC virus DNA sequences are frequently present in the human upper and lower gastrointestinal tract.
- Mad-1 is the exclusive JC virus strain present in the human colon, and its transcriptional control region has a deleted 98-base-pair sequence in colon cancer tissues.
- Genetic pathways in the evolution of morphologically distinct colorectal neoplasms.
- Colonic mucosal prostaglandin E2 and cyclooxygenase expression before and after low aspirin doses in subjects at high risk or at normal risk for colorectal cancer.
- Microsatellites in the eukaryotic DNA mismatch repair genes as modulators of evolutionary mutation rate.
- Specific inhibition of cyclooxygenase-2 (COX-2) expression by dietary curcumin in HT-29 human colon cancer cells.
- Oxidative stress increases frameshift mutations in human colorectal cancer cells.
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