Dr. Gordon Campbell Weir M.D.
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
Joslin Clinic One Joslin Place Boston MA, 02215About
Dr. Gordon Weir practices Endocrinology in Boston, MA. Dr. Weir specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Weir examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Harvard Med Sch, Boston Ma 1967
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Measurement of glucose concentrations in rats: differences between glucose meter and plasma laboratory results.
- Reversal of hyperglycemia in mice after subcutaneous transplantation of macroencapsulated islets.
- Islet transplantation restores normal levels of insulin receptor and substrate tyrosine phosphorylation and phosphatidylinositol 3-kinase activity in skeletal muscle and myocardium of streptozocin-induced diabetic rats.
- Glucose regulates expression of inositol 1,4,5-trisphosphate receptor isoforms in isolated rat pancreatic islets.
- Chronic hyperglycemia triggers loss of pancreatic beta cell differentiation in an animal model of diabetes.
- Prior streptozotocin treatment does not inhibit pancreas regeneration after 90% pancreatectomy in rats.
- Prolonged xenograft survival of islets infected with small doses of adenovirus expressing CTLA4Ig.
- In situ electrochemical oxygen generation with an immunoisolation device.
- Differentiation and expansion of beta cell mass in porcine neonatal pancreatic cell clusters transplanted into nude mice.
- Improved vascularization of planar membrane diffusion devices following continuous infusion of vascular endothelial growth factor.
- Beta-cell dysfunction in 48-hour glucose-infused rats is not a consequence of elevated plasma lipid or islet triglyceride levels.
- Effects of diabetes and hypoxia on gene markers of angiogenesis (HGF, cMET, uPA and uPAR, TGF-alpha, TGF-beta, bFGF and Vimentin) in cultured and transplanted rat islets.
- Islets in alginate macrobeads reverse diabetes despite minimal acute insulin secretory responses.
- Potential role of the early response gene c-myc in beta-cell adaptation to changes in glucose concentration.
- Beta-cell adaptation and decompensation during the progression of diabetes.
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