Dr. Daniel Richard Marsh MD
Physiatrist (Physical Medicine) | Pain Medicine
901 N Curtis Rd Suite 304 Boise ID, 83706About
Dr. Daniel Marsh is a physiatrist practicing in Boise, ID. Dr. Marsh is a medical doctor specializing in physical medicine and rehabilitation. As a physiatrist, Dr. Marsh focuses on a patients ability to function, and can treat multiple conditions that affect the brain, nerves, spine, bones, muscles, joints, ligaments and tendons. Dr. Marsh can diagnose and treat pain that is a result of injury, disease or a disabling condition. Physiatrists often lead a team of physical therapists, occupational therapists and physicians in a patients treatment or prevention plan.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The N-methyl-D-aspartate receptor splice variant NR1-4 C-terminal domain. Deletion analysis and role in subcellular distribution.
- Central mechanisms for autonomic dysreflexia after spinal cord injury.
- Neutralizing intraspinal nerve growth factor with a trkA-IgG fusion protein blocks the development of autonomic dysreflexia in a clip-compression model of spinal cord injury.
- Transient blockade of the CD11d/CD18 integrin reduces secondary damage after spinal cord injury, improving sensory, autonomic, and motor function.
- Autonomic dysreflexia, induced by noxious or innocuous stimulation, does not depend on changes in dorsal horn substance p.
- Comparison of effects of methylprednisolone and anti-CD11d antibody treatments on autonomic dysreflexia after spinal cord injury.
- Autonomic dysreflexia after spinal cord injury: central mechanisms and strategies for prevention.
- Methylprednisolone causes minimal improvement after spinal cord injury in rats, contrasting with benefits of an anti-integrin treatment.
- Loss of GABAergic interneurons in laminae I-III of the spinal cord dorsal horn contributes to reduced GABAergic tone and neuropathic pain after spinal cord injury.
- Role of polysialylated neural cell adhesion molecule in rapid eye movement sleep regulation in rats.
- Aging-associated oxidative stress modulates the acute inflammatory response in skeletal muscle after contusion injury.
- Ischaemia-reperfusion modulates inflammation and fibrosis of skeletal muscle after contusion injury.
- Development of autonomic dysreflexia after spinal cord injury is associated with a lack of serotonergic axons in the intermediolateral cell column.
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