Dr. Amr H Sawalha MD
Rheumatologist | Rheumatology
825 Ne 10th St Oupb 2300 Oklahoma City OK, 73104About
Dr. Amr Sawalha is a rheumatologist practicing in Ann Arbor, MI. Dr. Sawalha specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Sawalha treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Education and Training
Yarmouk Univ, Fac Med, (Jordan Univ Sci & Tech), Irbid, Jordan 1998
Jordan University of Science and Technology Faculty of Medicine 1998
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Ankylosing spondylitis in north Jordan: descriptive and analytical study.
- Genetic linkage of systemic lupus erythematosus with chromosome 11q14 (SLEH1) in African-American families stratified by a nucleolar antinuclear antibody pattern.
- Defective DNA methylation and CD70 overexpression in CD4+ T cells in MRL/lpr lupus-prone mice.
- Impaired T cell protein kinase C delta activation decreases ERK pathway signaling in idiopathic and hydralazine-induced lupus.
- Presence of anti-La autoantibody is associated with a lower risk of nephritis and seizures in lupus patients.
- Sulpha allergy in lupus patients: a clinical perspective.
- MEK/ERK pathway inhibitors as a treatment for inflammatory arthritis might result in the development of lupus: comment on the article by Thiel et al.
- Epigenetics and T-cell immunity.
- Defective T-cell ERK signaling induces interferon-regulated gene expression and overexpression of methylation-sensitive genes similar to lupus patients.
- Haemolytic anaemia in a multi-ethnic cohort of lupus patients: a clinical and serological perspective.
- Dehydroepiandrosterone in systemic lupus erythematosus.
- Uric acid directly promotes human T-cell activation.
- Epigenetic regulation and the pathogenesis of systemic lupus erythematosus.
- Variants within MECP2, a key transcription regulator, are associated with increased susceptibility to lupus and differential gene expression in patients with systemic lupus erythematosus.
- Aberrant DNA methylation in skin diseases.
Treatments
- Arthritis
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