Brian C Mansfield DPT
Physical Therapist
521 E Elder St Suite 106 Fallbrook CA, 92028About
Brian Mansfield is a physical therapist practicing in Fallbrook, CA. Brian Mansfield specializes in physical treatment to help a patient reduce pain, restore mobility, rehabilitate an injury, or increase movement and overall function. As a physical therapist, Brian Mansfield can treat multiple conditions with exercises, ultrasound, electrical stimulation, joint mobilization, heat, ice, massage, laser or light therapy and more. Brian Mansfield will create a treatment plan based on the patients specific injury or condition, and might target a specific body part or body system based on the individual.
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Expert Publications
Data provided by the National Library of Medicine- Pregnancy-specific glycoprotein gene expression in recurrent aborters: a potential correlation to interleukin-10 expression.
- Type I glycogen storage diseases: disorders of the glucose-6-phosphatase complex.
- The signature motif in human glucose-6-phosphate transporter is essential for microsomal transport of glucose-6-phosphate.
- Impaired glucose homeostasis, neutrophil trafficking and function in mice lacking the glucose-6-phosphate transporter.
- A glucose-6-phosphate hydrolase, widely expressed outside the liver, can explain age-dependent resolution of hypoglycemia in glycogen storage disease type Ia.
- The islet-specific glucose-6-phosphatase-related protein, implicated in diabetes, is a glycoprotein embedded in the endoplasmic reticulum membrane.
- A potential new role for muscle in blood glucose homeostasis.
- Brain contains a functional glucose-6-phosphatase complex capable of endogenous glucose production.
- cDNA sequence of the pregnancy-specific beta 1-glycoprotein-11s (PSG-11s).
- In islet-specific glucose-6-phosphatase-related protein, the beta cell antigenic sequence that is targeted in diabetes is not responsible for the loss of phosphohydrolase activity.
- Construction of a genomic DNA library of Mycobacterium paratuberculosis.
- Long-term correction of murine glycogen storage disease type Ia by recombinant adeno-associated virus-1-mediated gene transfer.
- Bone marrow-derived cells require a functional glucose 6-phosphate transporter for normal myeloid functions.
- Impaired neutrophil activity and increased susceptibility to bacterial infection in mice lacking glucose-6-phosphatase-beta.
- Gene therapy for type I glycogen storage diseases.
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