Dr. William J Koch M.D.
OB-GYN (Obstetrician-Gynecologist)
2773 Harris St Suite A Eureka CA, 95503About
Dr. William Koch is an obstetrician-gynecologist practicing in Eureka, CA. Dr. Koch specializes in women's health, particularly the female reproductive system, pregnancy and childbirth. As an obstetrician-gynecologist, or OB-GYN, Dr. Koch can treat a number of health issues related to the vagina, uterus, ovaries, fallopian tubes and breasts. Dr Koch can also treat women during pregnancy, labor, childbirth and the postpartum period. In this specialty, doctors focus on reproductive care from puberty through adulthood.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Beta2-adrenergic receptor overexpression in the developing mouse heart: evidence for targeted modulation of ion channels.
- Ontogeny of cardiac beta-adrenoceptor desensitization mechanisms: agonist treatment enhances receptor/G-protein transduction rather than eliciting uncoupling.
- Targeting Gbeta gamma signaling in arterial vascular smooth muscle proliferation: a novel strategy to limit restenosis.
- Molecular and functional mechanisms of right ventricular adaptation in chronic pulmonary hypertension.
- Enhanced contractility and decreased beta-adrenergic receptor kinase-1 in mice lacking endogenous norepinephrine and epinephrine.
- Treatment of major depression in the context of panic disorder.
- Molecular beta-adrenergic signaling abnormalities in failing rabbit hearts after infarction.
- Enhancement of cardiac function after adenoviral-mediated in vivo intracoronary beta2-adrenergic receptor gene delivery.
- Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression.
- L-type calcium current and contractility in ventricular myocytes from mice overexpressing the cardiac beta 2-adrenoceptor.
- In vivo inhibition of elevated myocardial beta-adrenergic receptor kinase activity in hybrid transgenic mice restores normal beta-adrenergic signaling and function.
- External support modulates G protein expression and receptor coupling in experimental vein grafts.
- Potential future therapies for heart failure: gene transfer of beta-adrenergic signaling components.
- Myocardial G protein-coupled receptor kinases: implications for heart failure therapy.
- Overexpression of the cardiac beta(2)-adrenergic receptor and expression of a beta-adrenergic receptor kinase-1 (betaARK1) inhibitor both increase myocardial contractility but have differential effects on susceptibility to ischemic injury.
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Eureka, CA 95503Get Direction
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