John Pearce Morrow MD
Cardiac Electrophysiologist | Clinical Cardiac Electrophysiology
630 W 168th St Ph Stem 10-410 New York NY, 10032About
Dr. John Morrow is a Cardiac Electrophysicist practicing in New York, NY. Dr. Morrow performs testing procedures on patients hearts electrical system in order to assess timing and abnormalities associated with electrical activities of the heart. Adult Cardiac Electrophysiologists are trained to treat irregular heartbeats, and carry out electrophysiology studies or ablations.
Education and Training
Columbia Univ Coll of Physicians And Surgeons, New York Ny 2000
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Ser1928 is a common site for Cav1.2 phosphorylation by protein kinase C isoforms.
- Activation of the BK (SLO1) potassium channel by mallotoxin.
- Potential application of late sodium current blockade in the treatment of heart failure and atrial fibrillation.
- Cardiac L-type calcium channel (Cav1.2) associates with gamma subunits.
- Diet-induced obesity causes long QT and reduces transcription of voltage-gated potassium channels.
- Enhanced efferocytosis of apoptotic cardiomyocytes through myeloid-epithelial-reproductive tyrosine kinase links acute inflammation resolution to cardiac repair after infarction.
- Extracellular signal-regulated kinase activation during cardiac hypertrophy reduces sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) transcription.
- Novel approaches to examine the regulation of voltage-gated calcium channels in the heart.
- Aberrant sodium influx causes cardiomyopathy and atrial fibrillation in mice.
- Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.
- Mitochondrial oxidative stress during cardiac lipid overload causes intracellular calcium leak and arrhythmia.
- Correction: Inhibition of NAPDH Oxidase 2 (NOX2) Prevents Oxidative Stress and Mitochondrial Abnormalities Caused by Saturated Fat in Cardiomyocytes.
- Inhibition of NADPH oxidase 2 (NOX2) prevents sepsis-induced cardiomyopathy by improving calcium handling and mitochondrial function.
- Acute CD47 Blockade During Ischemic Myocardial Reperfusion Enhances Phagocytosis-Associated Cardiac Repair.
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