Dr. Julie Kay Olson M.D,
Internist
1000 1st Dr Nw Austin MN, 55912About
Dr. Julie Olson, MD is one of the country's most highly rated doctors, specializing in internal medicine. MD Olson currently sees patients in Austin, Minnesota. Dr. Olson is a graduate of University Of ...
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Expert Publications
Data provided by the National Library of Medicine- Expression of functional NK-1 receptors in murine microglia.
- Epitope spreading and molecular mimicry as triggers of autoimmunity in the Theiler's virus-induced demyelinating disease model of multiple sclerosis.
- Innate and adaptive immune requirements for induction of autoimmune demyelinating disease by molecular mimicry.
- Mouse models of multiple sclerosis: experimental autoimmune encephalomyelitis and Theiler's virus-induced demyelinating disease.
- Microglia initiate central nervous system innate and adaptive immune responses through multiple TLRs.
- Differential activation of astrocytes by innate and adaptive immune stimuli.
- Initiation and exacerbation of autoimmune demyelination of the central nervous system via virus-induced molecular mimicry: implications for the pathogenesis of multiple sclerosis.
- The innate immune response affects the development of the autoimmune response in Theiler's virus-induced demyelinating disease.
- Innate immune CD11b+Gr-1+ cells, suppressor cells, affect the immune response during Theiler's virus-induced demyelinating disease.
- Immune response by microglia in the spinal cord.
- Activation of microglia depends on Na+/H+ exchange-mediated H+ homeostasis.
- Immune responses of microglia in the spinal cord: contribution to pain states.
- The Ifng gene is essential for Vdr gene expression and vitamin D₃-mediated reduction of the pathogenic T cell burden in the central nervous system in experimental autoimmune encephalomyelitis, a multiple sclerosis model.
- Endoplasmic reticulum stress regulates the innate immunity critical transcription factor IRF3.
- IFNγ influences type I interferon response and susceptibility to Theiler's virus-induced demyelinating disease.
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