Judith K Mickelson M.D.
Cardiologist | Cardiovascular Disease
3219 Clifton Ave Suite 400 Cincinnati OH, 45220About
Dr. Judith Mickelson is a cardiologist practicing in Cincinnati, OH. Dr. Mickelson specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Mickelson also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Baylor College of Medicine 1978
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Myocardial contrast echocardiography: relation between on-line and off-line assessment of myocardial perfusion.
- A monoclonal antibody against the platelet glycoprotein IIb/IIIa receptor complex prevents platelet aggregation and thrombosis in a canine model of coronary angioplasty.
- Prospective, double-blind, placebo-controlled trial of low-dose amiodarone in patients with severe heart failure and asymptomatic frequent ventricular ectopy.
- Free radical scavengers in myocardial ischemia.
- Sustained limitation by superoxide dismutase of canine myocardial injury due to regional ischemia followed by reperfusion.
- Protection of myocardial function and coronary vasculature by streptokinase.
- Identification of a time window for therapy to reduce experimental canine myocardial injury: suppression of neutrophil activation during 72 hours of reperfusion.
- Induction of ventricular arrhythmia by high and low osmolarity ionic and nonionic contrast media.
- Streptokinase improves reperfusion blood flow after coronary artery occlusion.
- Antiplatelet monoclonal F(ab')2 antibody directed against the platelet GPIIb/IIIa receptor complex prevents coronary artery thrombosis in the canine heart.
- Recombinant single-chain urokinase-type plasminogen activator (rscu-PA) induces thrombolysis and systemic fibrinolysis in a canine model of coronary artery thrombosis.
- Myocardial dysfunction and coronary vasoconstriction induced by
- Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody (anti-Mo1, anti-CD11b) that inhibits leukocyte adhesion.
- Dissolution of angiographically detected intracoronary thrombus for unstable angina pectoris after aspirin therapy.
- Electrolysis-induced myocardial dysfunction. A novel method for the study of free radical mediated tissue injury.
Treatments
- Heart Disease
- Heart Failure
- Coronary Artery Disease (cad)
- Pain
- Vascular Disease
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