Dr. Charles F Potter M.D.
Neonatal-Perinatal Medicine Specialist | Neonatal-Perinatal Medicine
415 N 9th St Springfield IL, 62702About
Dr. Charles Potter is a Neonatal-Perinatal Physician practicing in Springfield, IL. Dr. Potter cares for the critically ill newborn and premature infants. Neonatal-Perinatal Physicians treat conditions such as breathing disorders, birth defects, infections, and any other life-threatening medical problems. They coordinate with their young patients families and other physicians to determine appropriate treatment.
Board Certification
OtolaryngologyAmerican Board of OtolaryngologyABOto
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Absence of oroticaciduria in adenosine deaminase deficiency and purine nucleoside phosphorylase deficiency.
- Absence of oroticaciduria in adenosine deaminase deficiency and purine nucleoside phosphorylase deficiency.
- Purine metabolism and immunodeficiency: urinary purine excretion as a diagnostic screening test in adenosine deaminase and purine nucleoside phosphorylase deficiency.
- Purine metabolism and immunodeficiency: urinary purine excretion as a diagnostic screening test in adenosine deaminase and purine nucleoside phosphorylase deficiency.
- Purine metabolism in adenosine deaminase deficiency.
- Purine metabolism in adenosine deaminase deficiency.
- Complete deficiency of adenine phosphoribosyltransferase: a third case presenting as renal stones in a young child.
- Complete deficiency of adenine phosphoribosyltransferase: a third case presenting as renal stones in a young child.
- Hypertension, renal function and gout.
- Hypertension, renal function and gout.
- Purine and pyrimidine metabolism in hereditary oroticaciduria during a 15 year follow-up study.
- Purine and pyrimidine metabolism in hereditary oroticaciduria during a 15 year follow-up study.
- Evidence of a new syndrome involving hereditary uric acid over-production, neurological complications and deafness.
- Methylthioadenosine phosphorylase activity in human erythrocytes.
- Inosine formation from hypoxanthine by intact erythrocytes and fibroblasts of an immunodeficient child with purine nucleoside phosphorylase deficiency.
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