Dr. Stephen Jacob Pandol MD
Gastroenterologist | Gastroenterology
11301 Wilshire Blvd Los Angeles CA, 90073About
Dr. Stephen Pandol is a gastroenterologist practicing in Los Angeles, CA. Dr. Pandol specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Pandol performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Endoplasmic reticulum Ca(2+)-ATPase inhibitors stimulate membrane guanylate cyclase in pancreatic acinar cells.
- Multiple isoforms of the ryanodine receptor are expressed in rat pancreatic acinar cells.
- Cerulein upregulates ICAM-1 in pancreatic acinar cells, which mediates neutrophil adhesion to these cells.
- Activation of pancreatic acinar cells on isolation from tissue: cytokine upregulation via p38 MAP kinase.
- Localized pancreatic NF-kappaB activation and inflammatory response in taurocholate-induced pancreatitis.
- Ethanol metabolism and transcription factor activation in pancreatic acinar cells in rats.
- Caspase 8-mediated cleavage of plectin precedes F-actin breakdown in acinar cells during pancreatitis.
- Neutrophils and NADPH oxidase mediate intrapancreatic trypsin activation in murine experimental acute pancreatitis.
- Food-derived polyphenols inhibit pancreatic cancer growth through mitochondrial cytochrome C release and apoptosis.
- Cholecystokinin induces caspase activation and mitochondrial dysfunction in pancreatic acinar cells. Roles in cell injury processes of pancreatitis.
- Role of S-adenosylmethionine in two experimental models of pancreatitis.
- Curcumin ameliorates ethanol and nonethanol experimental pancreatitis.
- Emerging concepts for the mechanism of alcoholic pancreatitis from experimental models.
- Ethanol differentially regulates NF-kappaB activation in pancreatic acinar cells through calcium and protein kinase C pathways.
- Quantal calcium release and calcium entry in the pancreatic acinar cell.
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