Dr. Douglas V. Faller M.D.
Hematologist (Blood Specialist) | Hematology
732 Harrison Ave 3rd Floor Boston MA, 02118About
Dr. Douglas Faller is a hematology internist practicing in Boston, MA. Dr. Faller specializes in cases of suspected blood disorders when a diagnosis is unclear and further medical care is needed. Hematology internists work with other specialists such as surgeons, radiation therapists and gynecologists in order to get a better understanding on the cases. Often, patients are asked to participate in clinical research that could result in more efficient treatment methods.
Education and Training
Harvard Medical School 1979
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Hematology
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Endothelial cell responses to hypoxic stress.
- Sustained induction of fetal hemoglobin by pulse butyrate therapy in sickle cell disease.
- Establishment of order in the flow of genetic information in cells.
- Tryptophan concentrations in rat brain. Failure to correlate with free serum tryptophan or its ratio to the sum of other serum neutral amino acids.
- Feline leukemia virus long terminal repeat activates collagenase IV gene expression through AP-1.
- Selective inhibition of protein kinase C isozymes by Fas ligation.
- A novel DNA damage checkpoint involving post-transcriptional regulation of cyclin A expression.
- Acute reduction of brain serotonin and 5-HIAA following food consumption: correlation with the ratio of serum tryptophan to the sum of competing amino acids.
- Hypoxia affects tumor cell invasiveness in vitro: the role of hypoxia-activated ligand HAL1/13 (Ku86 autoantigen).
- DNA-damaging aryl hydrocarbons induce Mdm2 expression via p53-independent post-transcriptional mechanisms.
- Activation-induced nuclear translocation of RING3.
- RING3 kinase transactivates promoters of cell cycle regulatory genes through E2F.
- Oncogenic ras mediates apoptosis in response to protein kinase C inhibition through the generation of reactive oxygen species.
- Long terminal repeat regions from exogenous but not endogenous feline leukemia viruses transactivate cellular gene expression.
- Hypoxia-activated ligand HAL-1/13 is lupus autoantigen Ku80 and mediates lymphoid cell adhesion in vitro.
Fellowships
- Pediatric Hematology-Oncology, Children's Hospital, Boston MA , Hematology, Brigham and Women's Hospital, Boston MA 1984
- Dana Farber Cancer Institute, Fellow:Oncology 1983
- Brigham & Women's Hospital, Fellow:Hematology 1983
- Boston Children's Hospital, Fellow:Pediatric Hematology-Oncology 1983
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