Dr. Rahul Dnyaneshwar Pawar MBBS MD
Internist
Kumc Internal Medicine Medical 3901 Rainbow Blvd Ms Kansas City KS, 66160About
Dr. Rahul Pawar is an internist practicing in Kansas City, KS. Dr. Pawar specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Pawar provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- G-rich DNA suppresses systemic lupus.
- Molecular mechanisms of autoimmunity triggered by microbial infection.
- Toll-like receptor-7 modulates immune complex glomerulonephritis.
- Microbial nucleic acids pay a Toll in kidney disease.
- Expression and regulation of Toll-like receptors in lupus-like immune complex glomerulonephritis of MRL-Fas(lpr) mice.
- Ligands to nucleic acid-specific toll-like receptors and the onset of lupus nephritis.
- Inhibition of Toll-like receptor-7 (TLR-7) or TLR-7 plus TLR-9 attenuates glomerulonephritis and lung injury in experimental lupus.
- Coactivation of Toll-like receptor-3 and -7 in immune complex glomerulonephritis.
- Spiegelmer inhibition of CCL2/MCP-1 ameliorates lupus nephritis in MRL-(Fas)lpr mice.
- Requirement of Toll-like receptor 7 for pristane-induced production of autoantibodies and development of murine lupus nephritis.
- Molecular mimicry in innate immunity? The viral RNA recognition receptor TLR7 accelerates murine lupus.
- Viral 5'-triphosphate RNA and non-CpG DNA aggravate autoimmunity and lupus nephritis via distinct TLR-independent immune responses.
- Bacterial lipopeptide triggers massive albuminuria in murine lupus nephritis by activating Toll-like receptor 2 at the glomerular filtration barrier.
- Double-stranded RNA activates type I interferon secretion in glomerular endothelial cells via retinoic acid-inducible gene (RIG)-1.
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