Dr. Eric S Sobel MD
Rheumatologist | Rheumatology
1600 SW Archer Rd Gainesville FL, 32610About
Dr. Eric Sobel is a rheumatologist practicing in Gainesville, FL. Dr. Sobel specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Sobel treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Rheumatology
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Aberrant expression of the NF-kappaB and IkappaB proteins in B cells from viable motheaten mice.
- The major murine systemic lupus erythematosus susceptibility locus Sle1 results in abnormal functions of both B and T cells.
- Genetic dissection of systemic lupus erythematosus pathogenesis: evidence for functional expression of Sle3/5 by non-T cells.
- A comparison of the thyroidal plasma I131 clearance and the plasma protein-bound I131 tests for the diagnosis of hyperthyroidism.
- Some aspects of thyroidal iodine metabolism in a case of iodine-induced hypothyroidism.
- Effect of oral and intravenous cobaltous chloride on thyroid function.
- The role of CD5+ B cells in the production of autoantibodies in murine systemic lupus erythematosus.
- Mechanisms of peritoneal B-1a cells accumulation induced by murine lupus susceptibility locus Sle2.
- Genetic dissection of lupus pathogenesis: Sle3/5 impacts IgH CDR3 sequences, somatic mutations, and receptor editing.
- T cell hyperactivity in lupus as a consequence of hyperstimulatory antigen-presenting cells.
- Genetic dissection of systemic lupus erythematosus pathogenesis: partial functional complementation between Sle1 and Sle3/5 demonstrates requirement for intracellular coexpression for full phenotypic expression of lupus.
- High sensitivity C-reactive protein in systemic lupus erythematosus: relation to disease activity, clinical presentation and implications for cardiovascular risk.
- Comparison of chlordecone effects on autoimmunity in (NZBxNZW) F(1) and BALB/c mice.
- Acceleration of autoimmunity by organochlorine pesticides: a comparison of splenic B-cell effects of chlordecone and estradiol in (NZBxNZW)F1 mice.
- Diminished prolactin from chlordecone treatment in ovariectomized (NZBxNZW)F(1) mice.
Treatments
- Dermatomyositis, Rheumatoid Arthritis, Scleroderma And More
Fellowships
- University of North Carolina at Chapel Hill School of Medicine, Chapel Hill, NC 1990
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