Dr. John G. Wood
Dentist | General Practice
20100 N 51st Ave. Suite E-550 Glendale AZ, 85308About
Dr. John Wood is a Dentist practicing in Glendale, AZ. Dr. Wood specializes in preventing, diagnosing, and treating diseases and conditions associated with the mouth and overall dental health. Dentists are trained to carry out such treatment as professional cleaning, restorative, prosthodontic, and endodontic procedures, and performing examinations, among many others.
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Expert Publications
Data provided by the National Library of Medicine- Interaction between reactive oxygen species and nitric oxide in the microvascular response to systemic hypoxia.
- Mast cells mediate the microvascular inflammatory response to systemic hypoxia.
- Transgenic zebrafish model of neurodegeneration.
- Mesenteric microvascular inflammatory responses to systemic hypoxia are mediated by PAF and LTB4.
- Dissociation between skeletal muscle microvascular PO2 and hypoxia-induced microvascular inflammation.
- Activation of mast cells by systemic hypoxia, but not by local hypoxia, mediates increased leukocyte-endothelial adherence in cremaster venules.
- Exercise training prevents the inflammatory response to hypoxia in cremaster venules.
- Plasma from conscious hypoxic rats stimulates leukocyte-endothelial interactions in normoxic cremaster venules.
- Exposure to hypoxia results in uneven pulmonary blood flow distribution prior to pulmonary edema.
- Hypobaric hypoxia as a tool to study pregnancy-dependent responses at the maternal-fetal interface.
- Role of the renin-angiotensin system in the systemic microvascular inflammation of alveolar hypoxia.
- Alveolar macrophages are necessary for the systemic inflammation of acute alveolar hypoxia.
- The systemic inflammation of alveolar hypoxia is initiated by alveolar macrophage-borne mediator(s).
- Alveolar hypoxia, alveolar macrophages, and systemic inflammation.
- Fructose, but not dextrose, induces leukocyte adherence to the mesenteric venule of the rat by oxidative stress.
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Nearby Providers
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