Miss Monique B. Haviland MSPT, CSCS, PES, CPI
Physical Therapist
1667 Lucerne St Suite B Minden NV, 89423About
Monique Haviland is a physical therapist practicing in Minden, NV. Monique Haviland specializes in physical treatment to help a patient reduce pain, restore mobility, rehabilitate an injury, or increase movement and overall function. As a physical therapist, Monique Haviland can treat multiple conditions with exercises, ultrasound, electrical stimulation, joint mobilization, heat, ice, massage, laser or light therapy and more. Monique Haviland will create a treatment plan based on the patients specific injury or condition, and might target a specific body part or body system based on the individual.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Application of cladistics to the analysis of genotype-phenotype relationships.
- Biological complexity and strategies for finding DNA variations responsible for inter-individual variation in risk of a common chronic disease, coronary artery disease.
- Estimation of Hardy-Weinberg and pairwise disequilibrium in the apolipoprotein AI-CIII-AIV gene cluster.
- Expression of a new mutation (Axd) causing axial defects in mice correlates with maternal phenotype and age.
- Expression of the Axd (axial defects) mutation in the mouse is insensitive to retinoic acid at low dose.
- Cladistic analysis of the apolipoprotein AI-CIII-AIV gene cluster using a healthy French Canadian sample. I. Haploid analysis.
- Impact of apolipoprotein E genotype variation on means, variances, and correlations of plasma lipid, lipoprotein, and apolipoprotein traits in octogenarians.
- Genetic architecture of common multifactorial diseases.
- Association between common alleles of the low-density lipoprotein receptor gene region and interindividual variation in plasma lipid and apolipoprotein levels in a population-based sample from Rochester, Minnesota.
- Syndrome X: is it for real?
- Correlates of family history of coronary artery disease in children.
- The relationship between risk factor levels and presence of coronary artery calcification is dependent on apolipoprotein E genotype.
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