Dr. James E Posey M.D.
General Practitioner
801 CHURCH ST NE DECATUR AL, 35601About
Dr. James Posey is a general practitioner practicing in DECATUR, AL. Dr. Posey does not specialize in one area of medicine, however provides routine health care services. General practitioners typically have regular, even life-long patients who they provide health care services to. Dr. Posey provides services including physical exams, immunizations, and diagnosing and treating multiple illnesses and injuries. General practitioners typically work in private offices and clinics and have staffs of nurses and administators.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Borrelia oxidative stress response regulator, BosR: a distinctive Zn-dependent transcriptional activator.
- Mutations at embB codon 306 are an important molecular indicator of ethambutol resistance in Mycobacterium tuberculosis.
- Overexpression of the chromosomally encoded aminoglycoside acetyltransferase eis confers kanamycin resistance in Mycobacterium tuberculosis.
- Rapid detection of multidrug-resistant Mycobacterium tuberculosis by use of real-time PCR and high-resolution melt analysis.
- Bacillus Calmette-Guérin vaccination using a microneedle patch.
- Molecular detection of mutations associated with first- and second-line drug resistance compared with conventional drug susceptibility testing of Mycobacterium tuberculosis.
- Cellular immune responses to nine Mycobacterium tuberculosis vaccine candidates following intranasal vaccination.
- Independent large scale duplications in multiple M. tuberculosis lineages overlapping the same genomic region.
- New insights into fluoroquinolone resistance in Mycobacterium tuberculosis: functional genetic analysis of gyrA and gyrB mutations.
- Aminoglycoside cross-resistance in Mycobacterium tuberculosis due to mutations in the 5' untranslated region of whiB7.
- O-mannosylation of the Mycobacterium tuberculosis adhesin Apa is crucial for T cell antigenicity during infection but is expendable for protection.
- Antimycobacterial activity of DNA intercalator inhibitors of Mycobacterium tuberculosis primase DnaG.
- Disparities in capreomycin resistance levels associated with the rrs A1401G mutation in clinical isolates of Mycobacterium tuberculosis.
- Attrition of T-cell functions and simultaneous upregulation of inhibitory markers correspond with the waning of BCG-induced protection against tuberculosis in mice.
- Correlation between GyrA substitutions and ofloxacin, levofloxacin, and
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