Dr. Mohamed Hassan Sayegh MD
Nephrologist (Kidney Specialist) | Nephrology
75 Francis St Brigham And Women's Boston MA, 02115Rating
5/5
About
Dr. Mohamed Sayegh is a nephrologist practicing in Boston, MA. Dr. Sayegh specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Sayegh most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Govt. Dental College, Bangalore University, India 1984
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Inhibition of allorecognition by a human class II MHC-derived peptide through the induction of apoptosis.
- Blockade of CD40L/CD40 costimulatory pathway in a DST presensitization model of islet allograft leads to a state of Allo-Ag specific tolerance and permits subsequent engraftment of donor strain islet or heart allografts.
- Role of peripheral clonal deletion in tolerance induction with bone marrow transplantation and costimulatory blockade.
- Effect of hepatocyte growth factor on chronic rejection in rat renal allografts.
- Distinct tolerance pathways in sensitized rat recipients after blockade of activation signal 1 and signal 2.
- Developments in the clinical science of transplantation during the 20th century.
- Immunosuppressive strategies in transplantation.
- Immunomodulatory functions of hyaluronate in the LEW-to-F344 model of chronic cardiac allograft rejection.
- CTLA4 signals are required to optimally induce allograft tolerance with combined donor-specific transfusion and anti-CD154 monoclonal antibody treatment.
- Finally, CTLA4Ig graduates to the clinic.
- Immunomodulatory functions of low-molecular weight hyaluronate in an acute rat renal allograft rejection model.
- Costimulatory signal blockade in murine relapsing experimental autoimmune encephalomyelitis.
- Cellular and humoral mechanisms of vascularized allograft rejection induced by indirect recognition of donor MHC allopeptides.
- The role of B/T costimulatory signals in the immunopotentiating activity of neisserial porin.
- Distinct tolerance pathways in sensitized allograft recipients after selective blockade of activation signal 1 or signal 2.
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300 LONGWOOD AVENUE BOSTON MA 2115