James William Egan MD
Cardiologist | Cardiovascular Disease
920 Sunnyslope Rd Hollister CA, 95023About
Dr. James Egan is a cardiologist practicing in Hollister, CA. Dr. Egan specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Egan also practices preventative medicine, helping patients maintain a heart-healthy life.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Pioglitazone hydrochloride monotherapy improves glycemic control in the treatment of patients with type 2 diabetes: a 6-month randomized placebo-controlled dose-response study. The Pioglitazone 001 Study Group.
- Pioglitazone hydrochloride in combination with metformin in the treatment of type 2 diabetes mellitus: a randomized, placebo-controlled study. The Pioglitazone 027 Study Group.
- Pioglitazone hydrochloride in combination with sulfonylurea therapy improves glycemic control in patients with type 2 diabetes mellitus: a randomized, placebo-controlled study.
- Irradiation of liposarcoma.
- Irradiation of hemangiopericytoma of Stout.
- Identification of cardiac endothelin binding sites in rats: downregulation of left atrial endothelin binding sites in response to myocardial infarction.
- Primate vascular responses to octimibate, a non-prostanoid agonist at the
- Reduction of myocardial damage and polymorphonuclear leukocyte accumulation following coronary artery occlusion and reperfusion by the thromboxane receptor antagonist BM 13.505.
- Effect of propranolol on ischemic myocardial damage and left ventricular hypertrophy following permanent coronary artery occlusion or occlusion followed by reperfusion.
- Reduction of myocardial damage and polymorphonuclear leukocyte accumulation following coronary artery occlusion and reperfusion by the thromboxane receptor antagonist BM 13.505.
- BM 13.505, a selective thromboxane receptor antagonist, reduces myocardial infarct size following coronary artery reperfusion.
- Inhibition of peptidoleukotriene responses does not moderate ischemic damage or neutrophil infiltration in myocardium following coronary artery occlusion with reperfusion.
- Temporal relation between neutrophil accumulation and myocardial reperfusion injury.
- Method for quantification of myocardial infarction and inflammatory cell infiltration in rat cardiac tissue.
- Comparison of the effects of a thromboxane synthase inhibitor or prostacyclin in combination with a phosphodiesterase inhibitor for prevention of experimental thrombosis and sudden death in rabbits.
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