Jennifer Wagner-felker LCSW
Social Worker | Clinical
500 UNIVERSITY DR HERSHEY PA, 17033About
Jennifer Wagner-felker LCSW is a top Social Worker in HERSHEY, PA. With a passion for the field and an unwavering commitment to their specialty, Jennifer Wagner-felker LCSW is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Jennifer Wagner-felker LCSW is a prime example of a true leader in health care. As a leader and expert in their field, Jennifer Wagner-felker LCSW is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In HERSHEY, PA, Jennifer Wagner-felker LCSW is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Cardiolipin prevents rate-dependent uncoupling and provides osmotic stability in yeast mitochondria.
- Myo-inositol-1-phosphate (MIP) synthase: a possible new target for antibipolar drugs.
- Lithium and valproate decrease the membrane phosphatidylinositol/phosphatidylcholine ratio.
- Regulation of phosphatidylglycerophosphate synthase by inositol in Saccharomyces
- The effect of lithium on expression of genes for inositol biosynthetic enzymes in mouse hippocampus; a comparison with the yeast model.
- Valproate disrupts regulation of inositol responsive genes and alters regulation of phospholipid biosynthesis.
- Cardiolipin stabilizes respiratory chain supercomplexes.
- Aberrant cardiolipin metabolism in the yeast taz1 mutant: a model for Barth syndrome.
- Human 1-D-myo-inositol-3-phosphate synthase is functional in yeast.
- Absence of cardiolipin results in temperature sensitivity, respiratory defects, and mitochondrial DNA instability independent of pet56.
- Cardiolipin biosynthesis and mitochondrial respiratory chain function are interdependent.
- The human TAZ gene complements mitochondrial dysfunction in the yeast taz1Delta mutant. Implications for Barth syndrome.
- Post-translational regulation of phosphatidylglycerolphosphate synthase in response to inositol.
- Loss of function of KRE5 suppresses temperature sensitivity of mutants lacking mitochondrial anionic lipids.
- Valproate decreases inositol biosynthesis.
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