Michael G. Durniak MA, LPC
Counselor/Therapist | Professional
1022 N Trumbull St Bay City MI, 48708About
Michael Durniak is a counselor in Bay City, MI. Michael evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Counselors provide consultation for each patient and their families.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- The conformation of apolipoprotein A-I in high-density lipoproteins is influenced by core lipid composition and particle size: a surface plasmon resonance study.
- Evidence that phospholipids play a key role in pre-beta apoA-I formation and high-density lipoprotein remodeling.
- Apolipoprotein A-II inhibits high density lipoprotein remodeling and lipid-poor apolipoprotein A-I formation.
- Participation of macrophages in atherosclerotic lesion morphology in LDLr-/-
- Evidence that endothelial lipase remodels high density lipoproteins without mediating the dissociation of apolipoprotein A-I.
- Atheroprotective potential of macrophage-derived phospholipid transfer protein in
- The impact of glycation on apolipoprotein A-I structure and its ability to activate lecithin:cholesterol acyltransferase.
- Macrophage PLTP is atheroprotective in LDLr-deficient mice with systemic PLTP deficiency.
- Nonenzymatic glycation impairs the antiinflammatory properties of apolipoprotein A-I.
- Reduced cholesterol and triglycerides in mice with a mutation in Mia2, a liver protein that localizes to ER exit sites.
- Atherosclerosis induced by endogenous and exogenous toll-like receptor (TLR)1 or TLR6 agonists.
- Bone marrow-derived HL mitigates bone marrow-derived CETP-mediated decreases in HDL in mice globally deficient in HL and the LDLr.
- In vivo efficacy of HDL-like nanolipid particles containing multivalent peptide mimetics of apolipoprotein A-I.
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