Patricia A Grondin LCMHC, MS
Counselor/Therapist | Mental Health
229 COTTAGE ST LITTLETON NH, 03561About
Patricia Grondin is an Addiction Medicine Physician in LITTLETON, NH. Patricia evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Addiction Medicine Physicians provide medical care in addition to consultation for each patient and their families.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Shock-produced olivine glass: first observation.
- AMP-activated protein kinase is required for exercise-induced peroxisome proliferator-activated receptor co-activator 1 translocation to subsarcolemmal mitochondria in skeletal muscle.
- Over-expressing mitofusin-2 in healthy mature mammalian skeletal muscle does not alter mitochondrial bioenergetics.
- IL-6 is not essential for exercise-induced increases in glucose uptake.
- PIKfyve: a new fish in the growing pool of AMPK substrates.
- High-fat diet-induced mitochondrial biogenesis is regulated by
- AMPK phosphorylation of ACC2 is required for skeletal muscle fatty acid oxidation and insulin sensitivity in mice.
- Early oxidative shifts in mouse skeletal muscle morphology with high-fat diet consumption do not lead to functional improvements.
- Exercise-stimulated interleukin-15 is controlled by AMPK and regulates skin metabolism and aging.
- Skeletal muscle AMPK is essential for the maintenance of FNDC5 expression.
- AMPK deficiency in cardiac muscle results in dilated cardiomyopathy in the absence of changes in energy metabolism.
- Skeletal muscle ACC2 S212 phosphorylation is not required for the control of fatty acid oxidation during exercise.
- Extremely rapid increase in fatty acid transport and intramyocellular lipid accumulation but markedly delayed insulin resistance after high fat feeding in rats.
- The AMPK activator R419 improves exercise capacity and skeletal muscle insulin sensitivity in obese mice.
- Treatment of nonalcoholic fatty liver disease: role of AMPK.
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