Dr. Barry William Festoff M.D.
Neurologist | Neurology
4801 E Linwood Blvd Va Medical Center Kansas City MO, 64128About
Dr. Barry Festoff is a distinguished Neurologist in Kansas City, MO. Dr. Festoff specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Festoff employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Festoff is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Univ of Miami Sch of Med, Miami Fl 1966
University of Miami Leonard M. Miller School of Medicine 1966
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Insulin-like growth factor binding proteins in cerebrospinal fluid during human development and aging.
- Regulation of the dual function tissue transglutaminase/Galpha(h) during murine neuromuscular development: gene and enzyme isoform expression.
- Developmental regulation of amyloid precursor protein at the neuromuscular junction in mouse skeletal muscle.
- Selective developmental regulation of gene expression for insulin-like growth factor-binding proteins in mouse spinal cord.
- Intron-exon swapping of transglutaminase mRNA and neuronal Tau aggregation in Alzheimer's disease.
- Rapid upregulation of caspase-3 in rat spinal cord after injury: mRNA, protein, and cellular localization correlates with apoptotic cell death.
- Upregulation of neurotoxic serine proteases, prothrombin, and protease-activated receptor 1 early after spinal cord injury.
- Neuroprotective signal transduction in model motor neurons exposed to thrombin: G-protein modulation effects on neurite outgrowth, Ca(2+) mobilization, and apoptosis.
- Protein crosslinking, tissue transglutaminase, alternative splicing and neurodegeneration.
- Participation of protease-activated receptor-1 in thrombin-induced microglial activation.
- Persistent protease-activated receptor 4 signaling mediates thrombin-induced microglial activation.
- Thrombospondin, a platelet alpha-granule and matrix glycoprotein, is increased in muscle basement membrane of patients with amyotrophic lateral sclerosis.
- Prospects for the pharmacotherapy of amyotrophic lateral sclerosis : old strategies and new paradigms for the third millennium.
- In vitro studies of skeletal muscle membranes. Effects of denervation on the macromolecular components of cation transport in red and white skeletal muscle.
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