Anna Stoia CSFA
Physician Assistant | Surgical Technologist
4401 E. TUSCANY AVE. NAMPA ID, 83686About
Anna Stoia is a physician assistant practicing in NAMPA, ID. Anna specializes in preventing and treating human illness and injury by providing a broad range of care under the supervision of a physician. A physician assistants work can include physical exams, ordering and interpreting tests, performing procedures and prescribing medication. Often times, they can assist in surgery but the scope of practice can vary according to jurisdiction or health care setting.
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Expert Publications
Data provided by the National Library of Medicine- A novel ATP-diphosphohydrolase from human term placental mitochondria.
- An alternative model for the transmembrane segments of the yeast H+-ATPase.
- Differential response to chloroethylclonidine in blood vessels of normotensive and spontaneously hypertensive rats: role of alpha 1D- and alpha 1A-adrenoceptors in contraction.
- Modulation of 2-oxoglutarate dehydrogenase complex by inorganic phosphate, Mg(2+), and other effectors.
- Stalk segment 5 of the yeast plasma membrane H+-ATPase: mutational evidence for a role in glucose regulation.
- Kinetic characterization of the rotenone-insensitive internal NADH: ubiquinone oxidoreductase of mitochondria from Saccharomyces cerevisiae.
- Rapid purification and biochemical characterization of glucose kinase from Streptomyces peucetius var. caesius.
- Effect of calcium on mitochondria from human term placenta.
- Purification and characterization of glutathione reductase from Rhodospirillum rubrum.
- Interactions of calcium with yeast mitochondria.
- Inhibition of cAMP-dependent protein kinase A: a novel
- Induction of morphological changes in Ustilago maydis cells by octyl gallate.
- Functional properties of the Ustilago maydis alternative oxidase under oxidative stress conditions.
- α(1D)-Adrenoceptor regulates the vasopressor action of α(1A)-adrenoceptor in mesenteric vascular bed of α(1D)-adrenoceptor knockout mice.
- Captopril avoids hypertension, the increase in plasma angiotensin II but increases angiotensin 1-7 and angiotensin II-induced perfusion pressure in isolated kidney in SHR.
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