Sally Alejandra Soto B.A
Social Worker
2085 RUSTIN AVE RIVERSIDE CA, 92507About
Sally Alejandra Soto B.A is a top Social Worker in RIVERSIDE, CA. With a passion for the field and an unwavering commitment to their specialty, Sally Alejandra Soto B.A is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Sally Alejandra Soto B.A is a prime example of a true leader in health care. As a leader and expert in their field, Sally Alejandra Soto B.A is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In RIVERSIDE, CA, Sally Alejandra Soto B.A is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- A role for antibody and Fc receptor in the clearance of Brugia malayi microfilariae.
- Interferon-gamma and nitric oxide production are not required for the immune-mediated clearance of Brugia malayi microfilariae in mice.
- Eotaxin-1-regulated eosinophils have a critical role in innate immunity against experimental Brugia malayi infection.
- B cells play a regulatory role in mice infected with the L3 of Brugia pahangi.
- Highly homologous Mycobacterium tuberculosis chaperonin 60 proteins with differential CD14 dependencies stimulate cytokine production by human monocytes through cooperative activation of p38 and ERK1/2 mitogen-activated protein kinases.
- Mannose-binding lectin A-deficient mice have abrogated antigen-specific IgM
- Live Brugia malayi microfilariae inhibit transendothelial migration of neutrophils and monocytes.
- Eosinophils are important for protection, immunoregulation and pathology during infection with nematode microfilariae.
- C-reactive protein is essential for innate resistance to pneumococcal infection.
- Disorganization of the splenic microanatomy in ageing mice.
- Cystatin F Ensures Eosinophil Survival by Regulating Granule Biogenesis.
- Brugia malayi microfilariae adhere to human vascular endothelial cells in a C3-dependent manner.
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