Ms. Lydia Kathleen Diebolt
Social Worker
204 W 13TH ST LAWRENCE KS, 66044About
Ms. Lydia Kathleen Diebolt is a top Social Worker in LAWRENCE, KS. With a passion for the field and an unwavering commitment to their specialty, Ms. Lydia Kathleen Diebolt is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Ms. Lydia Kathleen Diebolt is a prime example of a true leader in health care. As a leader and expert in their field, Ms. Lydia Kathleen Diebolt is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In LAWRENCE, KS, Ms. Lydia Kathleen Diebolt is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor.
- The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site.
- Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus.
- Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment.
- Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures.
- Ethanol exposure and withdrawal sensitizes the rat hippocampal CA1 pyramidal cell region to beta-amyloid (25-35)-induced cytotoxicity: NMDA receptor involvement.
- A 24 h corticosterone exposure exacerbates excitotoxic insult in rat hippocampal slice cultures independently of glucocorticoid receptor activation or protein synthesis.
- Microtubule-associated targets in chlorpyrifos oxon hippocampal neurotoxicity.
- Methamphetamine exposure antagonizes N-methyl-D-aspartate receptor-mediated neurotoxicity in organotypic hippocampal slice cultures.
- Potentiation of N-methyl-D-aspartate receptor-mediated neuronal injury during methamphetamine withdrawal in vitro requires co-activation of IP3 receptors.
- Sex differences in the neurotoxic effects of adenosine A1 receptor antagonism during ethanol withdrawal: reversal with an A1 receptor agonist or an NMDA receptor antagonist.
- Exercise for depression: efficacy, safety and clinical trial implications.
- Intra-cornu ammonis 1 administration of the human immunodeficiency virus-1 protein trans-activator of transcription exacerbates the ethanol withdrawal syndrome in rodents and activates N-methyl-D-aspartate glutamate receptors to produce persisting sp
- Neurodegenerative effects of recombinant HIV-1 Tat(1-86) are associated with inhibition of microtubule formation and oxidative stress-related reductions in microtubule-associated protein-2(a,b).
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OLATHE MEDICAL CENTERl
20333 WEST 151ST STREET OLATHE KS 66061