Brenda L Kolinski MA, LLPC
Counselor/Therapist | Professional
527 COBB ST CADILLAC MI, 49601About
Brenda Kolinski is a counselor in CADILLAC, MI. Brenda evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Counselors provide consultation for each patient and their families.
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Expert Publications
Data provided by the National Library of Medicine- High cyclooxygenase 1 (COX-1) and cyclooxygenase 2 (COX-2) contents in mouse lung tumors.
- Butylated hydroxytoluene (BHT) induction of pulmonary inflammation: a role in tumor promotion.
- The lung tumor promoter, butylated hydroxytoluene (BHT), causes chronic inflammation in promotion-sensitive BALB/cByJ mice but not in promotion-resistant CXB4 mice.
- Quantitative trait locus mapping of susceptibilities to butylated hydroxytoluene-induced lung tumor promotion and pulmonary inflammation in CXB mice.
- Celecoxib reduces pulmonary inflammation but not lung tumorigenesis in mice.
- Genetic susceptibility to benzene-induced toxicity: role of NADPH: quinone oxidoreductase-1.
- Male mice deficient in microsomal epoxide hydrolase are not susceptible to benzene-induced toxicity.
- Variations in Prkdc and susceptibility to benzene-induced toxicity in mice.
- Susceptibility to neoplastic and non-neoplastic pulmonary diseases in mice: genetic similarities.
- Cytokines differentially regulate the synthesis of prostanoid and nitric oxide mediators in tumorigenic versus non-tumorigenic mouse lung epithelial cell lines.
- Signal transduction pathways of tumor necrosis factor--mediated lung injury induced by ozone in mice.
- Protection against inhaled oxidants through scavenging of oxidized lipids by macrophage receptors MARCO and SR-AI/II.
- Review paper: the role of inflammation in mouse pulmonary neoplasia.
- Transcriptomic analysis of pathways regulated by toll-like receptor 4 in a murine model of chronic pulmonary inflammation and carcinogenesis.
- Vanadium pentoxide induces pulmonary inflammation and tumor promotion in a strain-dependent manner.
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