Kimberly M Cavitt AUD
Audiologist-Hearing Aid Fitter
1420 W NORWOOD ST CHICAGO IL, 60660About
Kimberly M Cavitt AUD is a top Audiologist-Hearing Aid Fitter in CHICAGO, IL. With a passion for the field and an unwavering commitment to their specialty, Kimberly M Cavitt AUD is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Kimberly M Cavitt AUD is a prime example of a true leader in health care. As a leader and expert in their field, Kimberly M Cavitt AUD is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In CHICAGO, IL, Kimberly M Cavitt AUD is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Activation of the mitogen-activated protein kinase signaling pathway is instrumental in determining the ability of Mycobacterium avium to grow in murine macrophages.
- Mice lacking bioactive IL-12 can generate protective, antigen-specific cellular responses to mycobacterial infection only if the IL-12 p40 subunit is present.
- The study of Mycobacterium leprae infection in interferon-gamma gene--disrupted mice as a model to explore the immunopathologic spectrum of leprosy.
- IFN-gamma and NO in mycobacterial disease: new jobs for old hands.
- Immunological basis of the development of necrotic lesions following Mycobacterium avium infection.
- CD4 is required for the development of a protective granulomatous response to pulmonary tuberculosis.
- In vivo IL-10 production reactivates chronic pulmonary tuberculosis in C57BL/6 mice.
- Macrophage signalling upon mycobacterial infection: the MAP kinases lead the way.
- Role of chemokine ligand 2 in the protective response to early murine pulmonary tuberculosis.
- The cytolytic activity of natural killer cells is not involved in the restriction of Mycobacterium avium growth.
- A limited antigen-specific cellular response is sufficient for the early control of Mycobacterium tuberculosis in the lung but is insufficient for long-term survival.
- IL-27 signaling compromises control of bacterial growth in mycobacteria-infected mice.
- Disruption of granulocyte macrophage-colony stimulating factor production in the lungs severely affects the ability of mice to control Mycobacterium tuberculosis infection.
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METHODIST HOSPITAL OF CHICAGOl
5025 N PAULINA STREET CHICAGO IL 60640