Ms. Valerie Shaw LCSW
Social Worker | Clinical
703 Calvin Avery Dr Suite A West Memphis AR, 72301About
Ms. Valerie Shaw LCSW is a top Social Worker in MEMPHIS, TN. With a passion for the field and an unwavering commitment to their specialty, Ms. Valerie Shaw LCSW is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Ms. Valerie Shaw LCSW is a prime example of a true leader in health care. As a leader and expert in their field, Ms. Valerie Shaw LCSW is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In MEMPHIS, TN, Ms. Valerie Shaw LCSW is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Bi-directional modulation of T cell-dependent antibody production by prostaglandin E(2).
- Autoimmunity due to molecular mimicry as a cause of neurological disease.
- T cell receptors recognizing type II collagen in HLA-DR-transgenic mice characterized by highly restricted V beta usage.
- Arthritis in mice that are deficient in interleukin-1 receptor antagonist is dependent on genetic background.
- Quantitative trait loci, genes, and polymorphisms that regulate bone mineral density in mouse.
- Analysis of candidate genes of spontaneous arthritis in mice deficient for interleukin-1 receptor antagonist.
- Genomic dissection and prioritizing of candidate genes of QTL for regulating
- Dual effects of IL-1 overactivity on the immune system in a mouse model of arthritis due to deficiency of IL-1 receptor antagonist.
- Congenic mice provide evidence for a genetic locus that modulates spontaneous arthritis caused by deficiency of IL-1RA.
- Theranostic immunoliposomes for osteoarthritis.
- Effect of fluorosis on liver cells of VC deficient and wild type mice.
- Genomic locus on chromosome 1 regulates susceptibility to spontaneous arthritis in mice deficiency of IL-1RA.
- Detection of early cartilage damage using targeted nanosomes in a post-traumatic osteoarthritis mouse model.
- Non-invasive dual fluorescence in vivo imaging for detection of macrophage infiltration and matrix metalloproteinase (MMP) activity in inflammatory arthritic joints.
- Decreased expression levels of Ifi genes is associated to the increased resistance to spontaneous arthritis disease in mice deficiency of IL-1RA.
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