Barbara L Groening OTR
Occupational Therapist
8900 STATE LINE RD LEAWOOD KS, 66206About
Dr. Barbara Groening practices Occupational Medicine in LEAWOOD, KS. Dr. Groening evaluates the interaction between work and health. Occupational medicine physicians have general knowledge of worksite operations and are familiar with the toxic properties of materials used by employees and the potential hazards and stressors of work processes; in addition to being qualified to determine an employees physical and emotional fitness for work; diagnosing and treating occupational diseases; handling work related injuries; and having an understanding of rehabilitation methods, health education techniques, sanitation, and workers compensation laws.
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Expert Publications
Data provided by the National Library of Medicine- GPR105, a novel Gi/o-coupled UDP-glucose receptor expressed on brain glia and peripheral immune cells, is regulated by immunologic challenge: possible role in neuroimmune function.
- Detrimental deletions: mitochondria, aging and Parkinson's disease.
- Localization of Parkinson's disease-associated LRRK2 in normal and pathological human brain.
- The biology and pathobiology of LRRK2: implications for Parkinson's disease.
- Conditional transgenic mice expressing C-terminally truncated human alpha-synuclein (alphaSyn119) exhibit reduced striatal dopamine without loss of nigrostriatal pathway dopaminergic neurons.
- GTPase activity plays a key role in the pathobiology of LRRK2.
- A rat model of progressive nigral neurodegeneration induced by the Parkinson's disease-associated G2019S mutation in LRRK2.
- Genetic mouse models of neurodegenerative diseases.
- Localization of MAP1-LC3 in vulnerable neurons and Lewy bodies in brains of patients with dementia with Lewy bodies.
- Dopaminergic neuronal loss, reduced neurite complexity and autophagic abnormalities in transgenic mice expressing G2019S mutant LRRK2.
- Parkin promotes the ubiquitination and degradation of the mitochondrial fusion factor mitofusin 1.
- Mitochondrial dysfunction in genetic animal models of Parkinson's disease.
- Parkin reinvents itself to regulate fatty acid metabolism by tagging CD36.
- PARK9-associated ATP13A2 localizes to intracellular acidic vesicles and regulates cation homeostasis and neuronal integrity.
- α-Synuclein in central nervous system and from erythrocytes, mammalian cells, and Escherichia coli exists predominantly as disordered monomer.
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