John Balentine
Counselor/Therapist | Addiction (Substance Use Disorder)
6330 THORNTON AVE NEWARK CA, 94560About
John Balentine is an Addiction Medicine Counselor in NEWARK, CA. John evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Addiction Medicine counselors provide patient care and consultation for each patient and their families.
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Expert Publications
Data provided by the National Library of Medicine- Vaccines for cervical cancer.
- Wilms' tumour: connecting tumorigenesis and organ development in the kidney.
- Oncogenic activity of epidermal growth factor receptor kinase mutant alleles is enhanced by the T790M drug resistance mutation.
- The tumor suppressor WTX shuttles to the nucleus and modulates WT1 activity.
- A novel Wilms tumor 1 (WT1) target gene negatively regulates the WNT signaling pathway.
- The predominant WT1 isoform (+KTS) encodes a DNA-binding protein targeting the planar cell polarity gene Scribble in renal podocytes.
- Aberrant overexpression of satellite repeats in pancreatic and other epithelial cancers.
- The WTX tumor suppressor enhances p53 acetylation by CBP/p300.
- Inactivation of the tumor suppressor WTX in a subset of pediatric tumors.
- The IGF2 intronic miR-483 selectively enhances transcription from IGF2 fetal promoters and enhances tumorigenesis.
- EWS-FLI1 utilizes divergent chromatin remodeling mechanisms to directly activate or repress enhancer elements in Ewing sarcoma.
- The Ability to Diagnose Intrahepatic Cholangiocarcinoma Definitively Using Novel Branched DNA-Enhanced Albumin RNA In Situ Hybridization Technology.
- Erratum to: The Ability to Diagnose Intrahepatic Cholangiocarcinoma Definitively Using Novel Branched DNA-Enhanced Albumin RNA In Situ Hybridization Technology.
- Erratum to: The Ability to Diagnose Intrahepatic Cholangiocarcinoma Definitively Using Novel Branched DNA-Enhanced Albumin RNA In Situ Hybridization Technology.
- SIRT6 Suppresses Pancreatic Cancer through Control of Lin28b.
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