Melinda Lucille Randall LADC LSW
Counselor/Therapist | Addiction (Substance Use Disorder)
115 O ST SPARKS NV, 89431About
Melinda Randall is an Addiction Medicine Counselor in SPARKS, NV. Melinda evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Addiction Medicine counselors provide patient care and consultation for each patient and their families.
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Expert Publications
Data provided by the National Library of Medicine- Calcineurin triggers reactive/inflammatory processes in astrocytes and is upregulated in aging and Alzheimer's models.
- The Ca(V) 1.2 Ca(2+) channel is expressed in sarcolemma of type I and IIa myofibers of adult skeletal muscle.
- Hippocampal 'zipper' slice studies reveal a necessary role for calcineurin in the increased activity of L-type Ca(2+) channels with aging.
- Interleukin-1beta-dependent signaling between astrocytes and neurons depends critically on astrocytic calcineurin/NFAT activity.
- Recovery of afferent function and synaptic strength in hippocampal CA1 following traumatic brain injury.
- Cognitive decline in Alzheimer's disease is associated with selective changes in calcineurin/NFAT signaling.
- Disparate effects of serum on basal and evoked NFAT activity in primary astrocyte cultures.
- NFATs and Alzheimer's Disease.
- Proteolysis of calcineurin is increased in human hippocampus during mild cognitive impairment and is stimulated by oligomeric Abeta in primary cell culture.
- Disrupting function of FK506-binding protein 1b/12.6 induces the Ca²+-dysregulation aging phenotype in hippocampal neurons.
- Preparation of acute hippocampal slices from rats and transgenic mice for the study of synaptic alterations during aging and amyloid pathology.
- Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's disease.
- Inhibition of soluble tumor necrosis factor ameliorates synaptic alterations and Ca2+ dysregulation in aged rats.
- Targeting astrocytes ameliorates neurologic changes in a mouse model of Alzheimer's disease.
- Calcium dysregulation and neuroinflammation: discrete and integrated mechanisms for age-related synaptic dysfunction.
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