Ms. Sarah Greenberg LICENSED INDEPENDENT
Social Worker
1121 WASHINGTON ST SUITE 4 NEWTON MA, 02465About
Ms. Sarah Greenberg LICENSED INDEPENDENT is a top Social Worker in NEWTON, MA. With a passion for the field and an unwavering commitment to their specialty, Ms. Sarah Greenberg LICENSED INDEPENDENT is an expert in changing the lives of their patients for the better. Through their designated cause and expertise in the field, Ms. Sarah Greenberg LICENSED INDEPENDENT is a prime example of a true leader in health care. As a leader and expert in their field, Ms. Sarah Greenberg LICENSED INDEPENDENT is passionate about enhancing patient quality of life. They embody the values of communication, safety, and trust when dealing directly with patients. In NEWTON, MA, Ms. Sarah Greenberg LICENSED INDEPENDENT is a true asset to their field and dedicated to the profession of medicine.
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Expert Publications
Data provided by the National Library of Medicine- Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formation.
- Synthesis and secondary structural studies of penta(acetyl-Hmb)A beta(1-40).
- Histidine-13 is a crucial residue in the zinc ion-induced aggregation of the A beta peptide of Alzheimer's disease.
- The A beta 3-pyroglutamyl and 11-pyroglutamyl peptides found in senile plaque have greater beta-sheet forming and aggregation propensities in vitro than full-length A beta.
- The hydrophobic core sequence modulates the neurotoxic and secondary structure properties of the prion peptide 106-126.
- On-line high-performance liquid chromatography/mass spectrometric investigation of amyloid-beta peptide variants found in Alzheimer's disease.
- Substrate-bound beta-amyloid peptides inhibit cell adhesion and neurite outgrowth in primary neuronal cultures.
- Peptide thioester preparation by Fmoc solid phase peptide synthesis for use in native chemical ligation.
- A beta peptides and calcium influence secretion of the amyloid protein precursor from chick sympathetic neurons in culture.
- Amyloidogenicity and neurotoxicity of peptides corresponding to the helical regions of PrP(C).
- Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neurons.
- The Val-210-Ile pathogenic Creutzfeldt-Jakob disease mutation increases both the
- The amyloid-beta peptide and its role in Alzheimer's disease.
- Use of x-ray in the presence of an incorrect needle count.
- Improved preparation of amyloid-beta peptides using DBU as Nalpha-Fmoc deprotection reagent.
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Nearby Providers
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Mark Steinberg440 Humphrey Street, Suite 3 SWAMPSCOTT MA 01907
Nearest Hospitals
FRANCISCAN CHILDREN'S HOSPITAL & REHAB CENTERl
30 WARREN STREET BRIGHTON MA 2135