Cristie Lynn Barone RN
Nurse
1 CARMANS RD MASSAPEQUA PARK NY, 11762About
Cristie Barone is a nurse working in MASSAPEQUA PARK,NY. As a nurse, Cristie works in the healthcare sector, interacting directly with patients to ensure that they receive quality care, understand physician instructions, and have access to the services needed to maintain optimal health. Cristie holds an active license to practice, and continues to take an all-encompassing role in supporting a patient's wellbeing.
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Data provided by the National Library of Medicine- Monkey rotavirus binding to alpha2beta1 integrin requires the alpha2 I domain and is facilitated by the homologous beta1 subunit.
- Integrin-using rotaviruses bind alpha2beta1 integrin alpha2 I domain via VP4 DGE sequence and recognize alphaXbeta2 and alphaVbeta3 by using VP7 during cell entry.
- Effects on rotavirus cell binding and infection of monomeric and polymeric peptides containing alpha2beta1 and alphaxbeta2 integrin ligand sequences.
- Rotaviruses interact with alpha4beta7 and alpha4beta1 integrins by binding the same integrin domains as natural ligands.
- Rotavirus spike protein VP5* binds alpha2beta1 integrin on the cell surface and competes with virus for cell binding and infectivity.
- Rotavirus infection of infant and young adult nonobese diabetic mice involves extraintestinal spread and delays diabetes onset.
- Rotavirus infection accelerates type 1 diabetes in mice with established insulitis.
- In vivo effects of cytokines on pancreatic beta-cells in models of type I diabetes dependent on CD4(+) T lymphocytes.
- Interferon signalling in pancreatic beta cells.
- SOCS1 negatively regulates the production of Foxp3+ CD4+ T cells in the thymus.
- Determinants of the specificity of rotavirus interactions with the alpha2beta1 integrin.
- An indirect role for NK cells in a CD4(+) T-cell-dependent mouse model of type I
- Autoreactive cytotoxic T lymphocytes acquire higher expression of cytotoxic effector markers in the islets of NOD mice after priming in pancreatic lymph nodes.
- The pro-apoptotic BH3-only protein Bid is dispensable for development of insulitis and diabetes in the non-obese diabetic mouse.
- TNF receptor 1 deficiency increases regulatory T cell function in nonobese diabetic mice.
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