Dr. Michael J. Devito D.D.S,
Orthodontist
2745 State Road 580 Suite 103 Clearwater FL, 33761About
Dr. Michael Devito practices Orthodontics in Clearwater, FL. Comprehensive orthodontic treatment includes metal wires that are inserted into orthodontic brackets, which can be made from stainless steel or a more aesthetic ceramic material. The wires interact with the brackets to move teeth into desired positions. Being advanced in the field, Dr. Devito may also use Invisalign or other aligner trays that have been designed to align a patients? smile. As an orthodontist, Dr. Devito must recognize various characteristics of a malocclusion or dentofacial deformity, define the nature of the problem, including the etiology if possible, and design a treatment strategy based on the specific needs and desires of the patient.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Exposure assessment to dioxins from the use of tampons and diapers.
- Developmental exposure to brominated diphenyl ethers results in thyroid hormone disruption.
- Lack of antiandrogenic effects in adult male rats following acute exposure to 2,2-bis(4-chlorophenyl)-1,1-dichloroethylene (p,p'-DDE).
- Comparative responsiveness of hypothyroxinemia and hepatic enzyme induction in Long-Evans rats versus C57BL/6J mice exposed to TCDD-like and phenobarbital-like polychlorinated biphenyl congeners.
- EGF and TGF-alpha expression influence the developmental toxicity of TCDD: dose response and AhR phenotype in EGF, TGF-alpha, and EGF + TGF-alpha knockout mice.
- Physiologically based pharmacokinetic model for developmental exposures to TCDD in the rat.
- Accumulation of PBDE-47 in primary cultures of rat neocortical cells.
- Development of a refined database of mammalian relative potency estimates for dioxin-like compounds.
- Thyroid-hormone-disrupting chemicals: evidence for dose-dependent additivity or synergism.
- Comparison of the use of a physiologically based pharmacokinetic model and a classical pharmacokinetic model for dioxin exposure assessments.
- Use of a physiologically based pharmacokinetic model for rats to study the influence of body fat mass and induction of CYP1A2 on the pharmacokinetics of TCDD.
- Possible mechanisms of thyroid hormone disruption in mice by BDE 47, a major polybrominated diphenyl ether congener.
- Coordinated changes in xenobiotic metabolizing enzyme gene expression in aging male rats.
- In vitro metabolism of pyrethroid pesticides by rat and human hepatic microsomes and cytochrome p450 isoforms.
- Short-term exposure to triclosan decreases thyroxine in vivo via upregulation of hepatic catabolism in Young Long-Evans rats.
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2745 State Road 580 Suite 103 -Clearwater, FL 33761Get Direction
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