Dr. Marja M. Hurley MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
John Dempsey Hospital 263 Farmington Avenu Farmington CT, 06030About
Dr. Marja Hurley practices Endocrinology in Farmington, CT. Dr. Hurley specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Hurley examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ Of Ct Sch Of Med- Farmington Ct 1976
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Impaired osteoclast formation in bone marrow cultures of Fgf2 null mice in response to parathyroid hormone.
- Stat1 controls postnatal bone formation by regulating fibroblast growth factor signaling in osteoblasts.
- Prostaglandins differently regulate FGF-2 and FGF receptor expression and induce nuclear translocation in osteoblasts via MAPK kinase.
- PGF2alpha increases FGF-2 and FGFR2 trafficking in Py1a rat osteoblasts via clathrin independent and importin beta dependent pathway.
- University of Connecticut HPPI pipeline.
- Benzyl butyl phthalate influences actin distribution and cell proliferation in rat Py1a osteoblasts.
- Anti-apoptotic Bcl-2 enhancing requires FGF-2/FGF receptor 1 binding in mouse osteoblasts.
- Reduced expression and function of bone morphogenetic protein-2 in bones of Fgf2 null mice.
- Prostaglandin F2alpha involves heparan sulphate sugar chains and FGFRs to modulate osteoblast growth and differentiation.
- Exported 18-kDa isoform of fibroblast growth factor-2 is a critical determinant of bone mass in mice.
- Endogenous FGF-2 is critically important in PTH anabolic effects on bone.
- Fibroblast growth factor 2 positively regulates expression of activating transcription factor 4 in osteoblasts.
- Nuclear isoforms of fibroblast growth factor 2 are novel inducers of hypophosphatemia via modulation of FGF23 and KLOTHO.
- Signaling pathways implicated in PGF2alpha effects on Fgf2+/+ and Fgf2-/- osteoblasts.
- Disruption of the Fgf2 gene activates the adipogenic and suppresses the osteogenic program in mesenchymal marrow stromal stem cells.
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