Dr. Phillip D Holler MD
Dermatologist
3400 Civic Center Blvd. 1-330s Perelman Cent Philadelphia PA, 19104About
Dr. Phillip Holler is a dermatologist practicing in Philadelphia, PA. Dr. Holler specializes in skin care. Dermatologists evaluate and manage both common and uncommon skin conditions. These conditions include acne, psoriasis, warts, skin infections, atopic dermatitis, herpes simplex and more. Dermatologists are also experts in more complex skin diseases like impetigo, hidradenitis and milaria. Dr. Holler diagnoses skin problems and develops unique treatments plans for each individual patient.
Board Certification
DermatologyAmerican Board of DermatologyABD
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Bispecific agents target endogenous murine T cells against human tumor xenografts.
- In vitro evolution of a T cell receptor with high affinity for peptide/MHC.
- CD8(-) T cell transfectants that express a high affinity T cell receptor exhibit enhanced peptide-dependent activation.
- TCRs with high affinity for foreign pMHC show self-reactivity.
- A yeast display system for engineering functional peptide-MHC complexes.
- Quantitative analysis of the contribution of TCR/pepMHC affinity and CD8 to T cell activation.
- T cell receptors: affinities, cross-reactivities, and a conformer model.
- Targeting virus entry and membrane fusion through specific peptide/MHC complexes using a high-affinity T-cell receptor.
- High-affinity, peptide-specific T cell receptors can be generated by mutations in CDR1, CDR2 or CDR3.
- Cellular uptake followed by class I MHC presentation of some exogenous peptides contributes to T cell stimulatory capacity.
- The same genomic region conditions clonal deletion and clonal deviation to the CD8alphaalpha and regulatory T cell lineages in NOD versus C57BL/6 mice.
- Enhanced thymic selection of FoxP3+ regulatory T cells in the NOD mouse model of autoimmune diabetes.
- The impact of TCR-binding properties and antigen presentation format on T cell responsiveness.
- Retinoids putting the "a" in alopecia.
- Fgf9 from dermal gammadelta T cells induces hair follicle neogenesis after
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