Dr. Robert C Stanton M.D.
Nephrologist (Kidney Specialist) | Nephrology
1 Joslin Pl Renal Section, Josli Boston MA, 02215About
Dr. Robert Stanton is a nephrologist practicing in Boston, MA. Dr. Stanton specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Stanton most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Drexel University College of Medicine 1980
MCP HAHNEMANN UNIV (FORMERLY ALLEGHENY UNIV) 1980
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Nephrology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Importance of glucose-6-phosphate dehydrogenase activity in cell death.
- A method for determination of pyridine nucleotides using a single extract.
- High glucose inhibits glucose-6-phosphate dehydrogenase via cAMP in aortic endothelial cells.
- Glucose-6-phosphate dehydrogenase deficiency promotes endothelial oxidant stress and decreases endothelial nitric oxide bioavailability.
- Suppression of interleukin-1 beta-induced nitric oxide production in RINm5F cells by inhibition of glucose-6-phosphate dehydrogenase.
- Glucose-6-phosphate dehydrogenase overexpression decreases endothelial cell oxidant stress and increases bioavailable nitric oxide.
- Glucose-6-phosphate dehydrogenase modulates vascular endothelial growth factor-mediated angiogenesis.
- Diabetes causes inhibition of glucose-6-phosphate dehydrogenase via activation of PKA, which contributes to oxidative stress in rat kidney cortex.
- Aldosterone impairs vascular reactivity by decreasing glucose-6-phosphate dehydrogenase activity.
- Microangiopathy in older patients with diabetes mellitus.
- Does blood glucose control improve vascular outcomes in patients with type 2
- Glucose-6-phosphate dehydrogenase-deficient mice have increased renal oxidative stress and increased albuminuria.
- Early administration of enalapril and losartan to patients with type 1 diabetes.
- High glucose inhibits glucose-6-phosphate dehydrogenase, leading to increased oxidative stress and beta-cell apoptosis.
- High-normal serum uric acid increases risk of early progressive renal function loss in type 1 diabetes: results of a 6-year follow-up.
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