Dr. Michelle Anne Lowes MD PHD
Dermatologist
1230 York Ave New York NY, 10065About
Dr. Michelle Lowes is a dermatologist practicing in New York, NY. Dr. Lowes specializes in skin care. Dermatologists evaluate and manage both common and uncommon skin conditions. These conditions include acne, psoriasis, warts, skin infections, atopic dermatitis, herpes simplex and more. Dermatologists are also experts in more complex skin diseases like impetigo, hidradenitis and milaria. Dr. Lowes diagnoses skin problems and develops unique treatments plans for each individual patient.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Keratoacanthomas have an immunosuppressive cytokine environment of increased IL-10 and decreased GM-CSF compared to squamous cell carcinomas.
- Regression of melanoma, but not keratoacanthoma, is associated with increased HLA-B22 and decreased HLA-B27 and HLA-DR1.
- Cytokine profiles in spontaneously regressing basal cell carcinomas.
- Transient reactive papulotranslucent acrokeratoderma associated with cystic fibrosis.
- Efalizumab (anti-CD11a)-induced increase in peripheral blood leukocytes in psoriasis patients is preferentially mediated by altered trafficking of memory CD8+ T cells into lesional skin.
- Current concepts in the immunopathogenesis of psoriasis.
- Alefacept reduces infiltrating T cells, activated dendritic cells, and inflammatory genes in psoriasis vulgaris.
- TNF inhibition rapidly down-regulates multiple proinflammatory pathways in psoriasis plaques.
- Psoriasis vulgaris flare during efalizumab therapy does not preclude future use: a case series.
- Increase in TNF-alpha and inducible nitric oxide synthase-expressing dendritic cells in psoriasis and reduction with efalizumab (anti-CD11a).
- Prominent production of IL-20 by CD68+/CD11c+ myeloid-derived cells in psoriasis: Gene regulation and cellular effects.
- Pathogenesis and therapy of psoriasis.
- Eruptive papules during efalizumab (anti-CD11a) therapy of psoriasis vulgaris: a case series.
- Novel insight into the agonistic mechanism of alefacept in vivo: differentially expressed genes may serve as biomarkers of response in psoriasis patients.
- Alefacept (anti-CD2) causes a selective reduction in circulating effector memory T cells (Tem) and relative preservation of central memory T cells (Tcm) in psoriasis.
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Nearby Providers
- Prof. James Krueger MD, PHD1230 York Ave New York NY 10065
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